。 动脉高压不仅是心血管疾病(CVD)的危险因素 ， 而且还能影响斑块的稳定性 。 结果表明 ， 皮肤间充质干细胞通过抑制Th17细胞分化和调节巨噬细胞M2极化 ， 减轻血管紧张素Ⅱ诱导的高血压 。 这表明 ， 皮肤间充质干细胞不仅可以抑制动脉粥样硬化的形成 ， 而且在高血压的MSC治疗中也有潜在的作用 。 \">. Arterial hypertension is not only a cardiovascular disease (CVD) risk factor but can also influence plaque stability. It has been shown that skin-MSCs attenuated angiotensin II-induced hypertension by inhibiting Th17 cell differentiation and by modulating macrophage M2 polarization. This suggests that skin-MSCs do not only suppress the formation of the atherosclerosis but potentially also have a role in MSC based therapy for hypertension [\" newtip=\"
。 动脉高压不仅是心血管疾病(CVD)的危险因素 ， 而且还能影响斑块的稳定性 。 结果表明 ， 皮肤间充质干细胞通过抑制Th17细胞分化和调节巨噬细胞M2极化 ， 减轻血管紧张素Ⅱ诱导的高血压 。 这表明 ， 皮肤间充质干细胞不仅可以抑制动脉粥样硬化的形成 ， 而且在高血压的MSC治疗中也有潜在的作用 。 \"> 。 . Arterial hypertension is not only a cardiovascular disease (CVD) risk factor but can also influence plaque stability. It has been shown that skin-MSCs attenuated angiotensin II-induced hypertension by inhibiting Th17 cell differentiation and by modulating macrophage M2 polarization. This suggests that skin-MSCs do not only suppress the formation of the atherosclerosis but potentially also have a role in MSC based therapy for hypertension [\" newtip=\"
。 动脉高压不仅是心血管疾病(CVD)的危险因素 ， 而且还能影响斑块的稳定性 。 结果表明 ， 皮肤间充质干细胞通过抑制Th17细胞分化和调节巨噬细胞M2极化 ， 减轻血管紧张素Ⅱ诱导的高血压 。 这表明 ， 皮肤间充质干细胞不仅可以抑制动脉粥样硬化的形成 ， 而且在高血压的MSC治疗中也有潜在的作用 。 \">这表明 ， . Arterial hypertension is not only a cardiovascular disease (CVD) risk factor but can also influence plaque stability. It has been shown that skin-MSCs attenuated angiotensin II-induced hypertension by inhibiting Th17 cell differentiation and by modulating macrophage M2 polarization. This suggests that skin-MSCs do not only suppress the formation of the atherosclerosis but potentially also have a role in MSC based therapy for hypertension [\" newtip=\"
。 动脉高压不仅是心血管疾病(CVD)的危险因素 ， 而且还能影响斑块的稳定性 。 结果表明 ， 皮肤间充质干细胞通过抑制Th17细胞分化和调节巨噬细胞M2极化 ， 减轻血管紧张素Ⅱ诱导的高血压 。 这表明 ， 皮肤间充质干细胞不仅可以抑制动脉粥样硬化的形成 ， 而且在高血压的MSC治疗中也有潜在的作用 。 \">间. Arterial hypertension is not only a cardiovascular disease (CVD) risk factor but can also influence plaque stability. It has been shown that skin-MSCs attenuated angiotensin II-induced hypertension by inhibiting Th17 cell differentiation and by modulating macrophage M2 polarization. This suggests that skin-MSCs do not only suppress the formation of the atherosclerosis but potentially also have a role in MSC based therapy for hypertension [\" newtip=\"
。 动脉高压不仅是心血管疾病(CVD)的危险因素 ， 而且还能影响斑块的稳定性 。 结果表明 ， 皮肤间充质干细胞通过抑制Th17细胞分化和调节巨噬细胞M2极化 ， 减轻血管紧张素Ⅱ诱导的高血压 。 这表明 ， 皮肤间充质干细胞不仅可以抑制动脉粥样硬化的形成 ， 而且在高血压的MSC治疗中也有潜在的作用 。 \">充质干细胞不仅可以抑制动脉粥样硬化的形成 ， 而且在高血压的MSC治疗中也有潜在的作用
